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Correspondence |

Septic Facial Vein Thrombosis Due to Panton-Valentine Leukocidin–Positive Staphylococcus aureus

Norah Kramkimel, MD; Emilie Sbidian, MD; Tu Anh Duong, MD; Philippe Lesprit, MD; Jean-Claude Roujeau, PhD, MD; Martine Bagot, PhD, MD
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Copyright 2009 American Medical Association. All Rights Reserved. Applicable FARS/DFARS Restrictions Apply to Government Use.

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Arch Dermatol. 2009;145(12):1460-1461. doi:10.1001/archdermatol.2009.317
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Facial malignant staphylococcal infection is a rare infectious disorder characterized by a septic facial venous thrombosis. Herein we report the first case to our knowledge in a healthy young woman due to Panton-Valentine leukocidin (PVL)–positive Staphylococcus aureus.

REPORT OF A CASE

A 20-year-old woman with no medical history was admitted for facial edema with high fever. She had been treated with amoxicillin plus clavulanic acid for 48 hours without efficacy. For a few days prior to presentation, she had a handled intranasal pock. Physical examination revealed significant inflammatory facial edema with periorbital predominance (Figure 1) and bilateral cervical adenopathies. Neurological and ophthalmological findings were normal.

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Figure 1. Marked facial edema with predominance about the periorbital region, especially of the right eye (A) and total recovery after treatment (B).

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Laboratory tests showed leukocytosis (white blood cell count, 15 200/μL [reference range, 4000-10 000/μL]) and an increased C-reactive protein serum concentration (170 mg/L [reference, <5 mg/L]). (To convert white blood cells to number of cells × 109 per liter, multiply by 0.001; to convert C-reactive protein to nanomoles per liter, multiply by 9.524.) Blood cultures and nasal swabs grew gram-positive cocci, which were identified as PVL-positive, methicillin-sensitive S aureus.

On computed tomography (CT), extended facial cellulitis was found with facial veins and right superior ophthalmic vein thrombosis (Figure 2). A transthoracic echocardiogram showed no abnormalities. The diagnosis of malignant staphylococcal infection of the face was confirmed. Intravenous penicillin M (100 mg/kg/d) was given for 3 weeks in combination with gentamicin (3 mg/kg/d) for the first 48 hours. Effective anticoagulation was maintained for 6 months until the venous thrombosis had completely disappeared.

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Figure 2. Computed tomography images. A and B, Important infiltration of facial soft tissues and filling defects of the bilateral facial veins (arrow). C, Filling defects of the right superior ophthalmic vein (left arrow); the left superior ophthalmic vein is permeable (right arrow).

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COMMENT

Malignant staphylococcal infection of the face is a rare but life-threatening Staphylococcus aureus septicemia. The death rate is around 20%.1 It corresponds to a septic facial vein thrombosis and commonly follows facial furuncle handling. The causal lesion is not always defined, as it was not in our patient. Onset is sudden and includes clinical status deterioration, high fever, and chills. Clinical examination shows an inflammatory purpuric facial edema with fast-spreading and severe pain. On account of cerebral venous thrombosis, careful neurologic and ophthalmologic examination is essential. The main complication is cavernous sinus thrombosis.

In the present case, the painful periorbital edema with fever and the ineffective 2-day large-dose antibiotic therapy prompted physicians to perform a CT scan looking for septic facial thrombosis. Magnetic resonance imaging is currently the gold standard for septic venous thrombosis diagnosis. Initially, thrombus is T2 hypointense and then T1 and T2 hyperintense1 with sinus broadening.

Our case is remarkable because it involved a virulent, community-acquired PVL-positive, methicillin-sensitive S aureus skin infection in a healthy young woman. Panton-Valentine leukocidin S aureus is responsible for recent outbreaks of severe invasive infections in young patients without substantial predisposing risk factors.2 In the United Kingdom, 4.9% of S aureus clinical diseases are PVL positive.3 Panton-Valentine leukocidin–positive S aureus is strongly associated with skin and soft-tissue infections such as abscesses, skin lesions, and boils (P < .001), but it is not statistically significantly associated with impetigo, blisters, and staphylococcal scalded skin syndrome.3

With regard to the thrombosis care, we did not perform thrombophilia exploration. Invasion of endothelial cells by S aureus is responsible for septic thrombosis.4

In conclusion, severe facial infection (painful palpebral edema with high fever, clinical state deterioration, and neurologic and ophthalmologic symptoms) must raise the diagnostic possibility of facial malignant staphylococcal infection. Intravenous antibiotic therapy combined with anticoagulation therapy must be started as early as possible.

AUTHOR INFORMATION

Correspondence: Dr Bagot, Service de Dermatologie, Hôpital Henri Mondor, 51 avenue Maréchal de Lattre de Tassigny, 94010 Créteil, France (martine.bagot@hmn.aphp.fr).

Financial Disclosure: None reported.

REFERENCES

Ayadi  K, Mehdi  M, Khannous  M.  et al.  Septic thrombosis of the cavernous sinus complicating malignant staphylococcal infection of the face. J Radiol 1995;76 (8) 497- 499 7473387
Gillet  Y, Issartel  B, Vanhems  P.  et al.  Association between Staphylococcus aureus strains carrying gene for Panton-Valentine leukocidin and highly lethal necrotising pneumonia in young immunocompetent patients. Lancet 2002;359 (9308) 753- 759 11888586
Holmes  A, Ganner  M, McGuane  S, Pitt  TL, Cookson  BD, Kearns  AM. Staphylococcus aureus isolates carrying Panton-Valentine leucocidin genes in England and Wales: frequency, characterization, and association with clinical disease. J Clin Microbiol 2005;43 (5) 2384- 2390 15872271
Sinha  B, Herrmann  M. Mechanism and consequences of invasion of endothelial cells by Staphylococcus aureus. Thromb Haemost 2005;94 (2) 266- 277 16113815

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Figures

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Figure 1. Marked facial edema with predominance about the periorbital region, especially of the right eye (A) and total recovery after treatment (B).

Grahic Jump Location
Place holder to copy figure label and caption

Figure 2. Computed tomography images. A and B, Important infiltration of facial soft tissues and filling defects of the bilateral facial veins (arrow). C, Filling defects of the right superior ophthalmic vein (left arrow); the left superior ophthalmic vein is permeable (right arrow).

Grahic Jump Location

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Ayadi  K, Mehdi  M, Khannous  M.  et al.  Septic thrombosis of the cavernous sinus complicating malignant staphylococcal infection of the face. J Radiol 1995;76 (8) 497- 499 7473387
Gillet  Y, Issartel  B, Vanhems  P.  et al.  Association between Staphylococcus aureus strains carrying gene for Panton-Valentine leukocidin and highly lethal necrotising pneumonia in young immunocompetent patients. Lancet 2002;359 (9308) 753- 759 11888586
Holmes  A, Ganner  M, McGuane  S, Pitt  TL, Cookson  BD, Kearns  AM. Staphylococcus aureus isolates carrying Panton-Valentine leucocidin genes in England and Wales: frequency, characterization, and association with clinical disease. J Clin Microbiol 2005;43 (5) 2384- 2390 15872271
Sinha  B, Herrmann  M. Mechanism and consequences of invasion of endothelial cells by Staphylococcus aureus. Thromb Haemost 2005;94 (2) 266- 277 16113815

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