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Correspondence |

Mondor Disease in a Patient With Psoriasis Treated With an Anti–Interleukin 12/Interleukin 23 Investigational Drug

Stefanie A. Hirano, MD; Ashley R. Mason, MD; Molly M. Warthan, MD; Robert J. Pariser, MD
Arch Dermatol. 2010;146(9):1049-1050. doi:10.1001/archdermatol.2010.232.
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Mayor  MBurón  Ide Mora  JC  et al.  Mondor's disease. Int J Dermatol 2000;39 (12) 922- 925
PubMed Link to Article[[XSLOpenURL/10.1046/j.1365-4362.2000.00018.x]]
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PubMed Link to Article[[XSLOpenURL/10.1084/jem.20030896]]
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PubMed Link to Article[[XSLOpenURL/10.1038/nri1648]]
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PubMed Link to Article[[XSLOpenURL/10.1084/jem.180.3.907]]
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PubMed Link to Article[[XSLOpenURL/10.1055/s-0029-1222610]]
Karabudak  OUlusoy  REErikci  AASolmazgul  EDogan  BHarmanyeri  Y Inflammation and hypercoagulable state in adult psoriatic men. Acta Derm Venereol 2008;88 (4) 337- 340
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PubMed Link to Article[[XSLOpenURL/10.1001/jama.296.14.1735]]
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Figure 1.

Initial presentation. Skin retraction on the anterolateral chest wall is observed on arm abduction. The indurated, longitudinal cordlike bands are consistent with clinical findings of Mondor disease. Skin around the cords appears indented.

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Figure 2.

The biopsy specimen from the third episode of Mondor disease showed venous thrombosis in the deep dermis with some evidence of recanalization. A thickened venous wall and lymphocytic infiltrate were also observed. Findings were consistent with superficial thrombophlebitis (hematoxylin-eosin, original magnification ×100).

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